ON-FIELD PHYSIOTHERAPY
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ON-FIELD PHYSIOTHERAPY
CONTENTS Roles
of on-field physiotherapist Basic assessment procedures Prevention and assessment of heat & cold injuries Management of acute soft tissue injuries
Why we need on field physiotherapy?
ON FIELD PHYSIOTHERAPIST Roles
On-field services in Hong Kong Team Physiotherapist Domestic, National and International Level
ON-FIELD PHYSIOTHERAPY SERVICES 1996
International Masters Hockey Tournament 1996 Seoul International Women’s Road relay Hong Kong Cricket team - Bangladesh Cricket Tournament Standard Chartered Shenzen - Hong Kong Marathon
Swire
Group Tsing Ma Bridge 10 km and Marathon Hong Kong - Beijing Relay Standard Chartered International Marathon (1997-2002)
Sports Physiotherapy in Elite level SDB,
5 full time physiotherapists Hong Kong Team physiotherapists 1987 Asian Athletic Championship 1992 Barcelona Olympics, 1994 Commonwealth and Asian Games, 1996 Altanta Olympics (3) 2002 Busan Asian Games: 8 physiotherapists
KNOW THE SPORT
ON FIELD SERVICES KNOW THE VENUE
PREPARATION Deployment Equipment
of manpower
Types of injury
Cases
Muscle soreness/strain
175
Ligamentous sprain
112
Tendonitis
44
Joint problems
41
Contusion
42
Laceration
7
Haematoma
3
Concussion
1
Others
6
Types of services
Number
IFT
44
US
158
TENS
34
HVG
7
ICE
98
Manual technique
210
Massage
211
Strapping and taping
70
Dressing
13
Education and advice
85
ON-FIELD MANAGEMENT ASSESSMENT
VENUE
PERSON
PROCEDURES
SYSTEMATIC APPROACH
Scene Survey
Is the scene safe?
It
is frequently better to remain uncertain about a diagnosis and feel mildly folish than to be constantly certain and confirm that you are an absolute fool.
PRIMARY SURVEY OF THE PATIENT LOC
Talk to the patient and assess his level of consciousness A Alert V Response to vocal stimuli P Response to pain U Unresponsive
AIRWAY + C-SPINE CONTROL
Is the victim able to maintain his airway If he can talk, the airway is OK Open airway by jaw thrust if necessary Do not use head tilt as this may affect the C-spine
BREATHING
Assess if victim is breathing adequately. Is it too fast? too slow? too shallow?
Oxygen, if available, should be given if breathing is laboured.
Feel for any tenderness
BREATHING
Auscultate the chest for unequal air entry
Check if the trachea is central
CIRCULATION Arrest any visible haemorrhage using direct pressure Check both carotid and radial pulse If radial pulse is weak or not palpable, the patient is probably in shock Capillary refill is less than 2 second normally
CIRCULATION If the patient is unresponsive and with
no carotid pulse==> this is cardiac arrest. you should start cardiopulmonary resuscitation immediately
DECISION POINT : SEND FOR THE AMBULANCE IMMEDIATELY Impaired
conscious state Airway obstruction Breathing difficulties Significant external bleeding especially when control by external pressure is ineffective
DECISION POINT : SEND FOR THE AMBULANCE IMMEDIATELY feature
of shock: thready pulse, cold clammy hands, delayed capillary refill unstable pelvis major fracture of limb bones
CARDIOPULMONARY ARREST PROBABLE
CAUSES: HEAD TRAUMA Cx INJURY MAXILOFACIAL OR THORACIC TRAUMA CVA MYOCARDIAL INFARCTION
HAEMORRHAGE INTERNAL: COLD RAPID
PULSE AND RESPIRATION PALPABLE PAIN AND TENDERNESS RESTLESSNESS EXCESSIVE THIRST BLOOD IN THE URINE OR STOOL OBSERVE FOR SHOCK OR ARREST
HAEMORRHAGE External Direct Pressure Arterial Pressure Pt. Compression Area should be elevated
SECONDARY ASSESSMENT Chief
Complaints Behaviour of symptoms Location & radiation of the symptoms Mode of onset Mechanism of injury Functional alterations Related symptoms Past injuries
LOOK AND PALPATION Location
of Pain Degree & type of swelling Temperature & texture of the area Muscle spasm Tissue continuity & deformity Neuromuscular function Abnormal Motion or sensation
MOVEMENT Active
& Functional Motions Resistive Motion Specific Stress Test Sport Specific Function Return to activity
HEAT INJURY
Metabolic Heat Stress Metabolic heat production Exercise Shivering Thryoxine Sympathetic stimulation
Exercise 20-25x 25% efficiency
Heat Production
Heat Balance Radiation Conduction Convection Evaporation
Heat loss
WBGT 0.1: 0.7: 0.2
CONVECTION Responsible
for transferring heat from working muscles and the skin surface Temp differential between skin and environment Heat transfer coefficient, body surface area and wind velocity Minimal body fat and loose-fitting clothing
CONDUCTION Minimal
effect on body heat transfer Direct contact between skin and an object
RADIATION Solar
radiation and radiation from tracks, roads, and surrounding structures Can be a major contributor to heat load
EVAPORATION Most
important heat dissipation mechanism in warm environments Sweating – a fit athlete can produce up to 30 ml of sweat per min Evaporation depends evaporative heat transfer coefficient – air velocity and water vapor pressure gradient (relative humidity)
WBGT Wet
Bulb Globe Temperature Three monitors: Dry bulb (Tdb) air temperature Wet bulb (Twb) relative humidity Black globe (Tg) solar radiation WBGT = 0.1Tdb + 0.7Twb + 0.2Tg
Without adaptive mechanisms, moderate exercise could elevate temp by 1C every 5-6’
Fluid/electrolyte Loss of solutions Sodium andadded Prevention: Potassium salt to food, high K+ diet
HEAT INJURY
Heat Cramp
Warm, humid conditions, inadequate Cool fluids fluid pre-hydrate replacement
Dehydration
Red, Hot and Dry skin Heat exhaustion Strong & Rapid pulse LackMedical of sweating, CNS symptoms unsteady Emergency !! gait Heat Stroke confusion, combative behaviour, coma
Profuse Sweating Clammy & Cool Shading Skin remove excess Headache &cloth cooling with ice, Weakness sponges Nausea & hydration Weakness monitor Rapid vital Pulsesign & hospital Disorientation
PREVENTION Conditioning
sweat rate Acclimatization 3-4 Thirst: hrs/day, poor indicator 60-70% load core temperature Intake: 400-600 ml 15-20’ 5-10 days volume Fluid replacement 2-3%; plasma 200-300 ml every 15-20’ exercise heat storage Venue and schedule intense 3L/hr Every L loss, 0.3 C Q 1L/min HR 8
CHILDREN AND HEAT INJURY
Sweat less effectively; produce metabolic heat for given workload; acclimatize more slowly than adults; larger M/A; renal tubular filtration rate; self perceive;
BUT HOW ABOUT COLD INJURY? Heat
loss also depends on air movement, humidity, evaporation (sweating) and ambient temperature Wind velocity exacerbates heat losses
Adequate clothing High energy bar Avoid wind exposure
Medical Emergency !!
Mild Hypothermina
Moderate Hypothermia
Severe Hypothermia
Shivering, cold, hunger Confusion muscle spasm Slow pace Semi-conscious confused actions Extremely tired Poor coordination Muscle stiffness Slurred speech Disorientation Loss of consciousness Faint heartbeat
Acute Sports Injuries Treatment that comes with PRICE!
MANAGEMENT OF ACUTE SOFT TISSUE INJURIES PRICE
HARM
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